关于 "Cytoskeleton & Motor Proteins" 的结果237+ 个结果

CEF14, EBV Rta Protein (28-37) 来自 Epstein-Barr 病毒 Rta 蛋白的 HLA A24 限制性表位。

C-Reactive Protein (CRP) 201-206 是 C-反应蛋白的 201-206 片段。C-Reactive Protein 可作为炎症的标记，是心血管风险的标志物，可能促进动脉粥样硬化形成。

C-Reactive Protein (CRP) 77-82 是 C-反应蛋白的 77-82 片段。C-Reactive Protein 可作为炎症的标记，是心血管风险的标志物，可能促进动脉粥样硬化形成。

Interphotoreceptor Retinoid Binding Protein Fragment (IRBP)，具有 20 个残基肽，也是主要的致病表位，存在于光受体间类视黄醇结合蛋白肽 (IRBP 161-180) 的第一个同源重复中, 可引起后葡萄膜炎 (EAU)。

Interphotoreceptor retinoid-binding protein(668-687) 是光受体视黄醇类结合蛋白的 668 到 687 氨基酸序列，可诱发葡萄膜炎。

H13R, a mutation in the metal-binding region of Abeta reduces its copper-mediated toxicity. The native rodent sequence containing an arginine at this position is more tolerant to metals than the human amyloid peptide.

The English (H6R) mutation of β-amyloid peptides accelerates fibrillation without increasing protofibril formation. Ono et al. showed that the English and Tottori mutations alter Abeta assembly at its earliest stages, monomer folding and oligomerization, and produce oligomers that are more toxic to cultured neuronal cells than are wild type oligomers. The exchange of His? by Arg influences the structure of the Cu(II) complex formed by Aβ peptides.

The Iowa (D23N) mutant of Aβ 40 considerably more rapidly assembles in solution to form fibrils than the WT Aβ sequence. These fibrils also show a different structure, which could be responsible for their increased toxicity.

The Tottori (D7N) mutation of β-amyloid peptides accelerates fibrillation without increasing protofibril formation. Ono et al. showed that the English and Tottori mutations alter Abeta assembly at its earliest stages, monomer folding and oligomerization, and produce oligomers that are more toxic to cultured neuronal cells than are wild type oligomers.

The G37D mutant does not show the aggregation behavior of WT Abeta42 nor its neurotoxicity.

Cys-Aβ1-40 can be easily and selectively modified, labeled, coupled to carriers e.g. by maleimide chemistry without affecting the sequences involved in fibril formation. The free mercapto moiety of the peptide adheres to gold surfaces.

Aβ40 S26C has been used for generating the covalently linked Aβ40 homodimer. Dimerization can be easily reverted by reducing the soluble dimer with thiols as β-mercaptoethanol. Aβ40 S26C is perfectly suited for labeling with fluorescent tags

A prion peptide

Myelin Basic Protein(87-99) TFA 是一种致脑肽，可诱导碱性蛋白特异性 T 细胞增殖。Myelin Basic Protein(87-99) TFA 在外周血单核细胞中引起 Th1 极化，与多发性硬化症 (MS) 有关。

组织因子 (TF) 通过与循环因子 VII 或 VIIa 形成复合物来启动血液凝固，从而形成 [TF:VIIa] 复合物。

Protein deglycase DJ-1 against-1 是 DJ-1 结合化合物，依赖地靶向 DJ1。Protein deglycase DJ-1 against-1 可以穿透血脑屏障 (BBB)。Protein deglycase DJ-1 against-1 是神经保护剂，具有用于帕金森病研究的潜力。

HIV-1 TAR RNA-binding protein

Agouti-related peptide(25-82)

Signal transducing molecules

PD-1（程序性细胞死亡 1）蛋白负向调节免疫反应并影响细胞凋亡和耐受诱导等过程。

[Ala107]-Myelin basic protein (MBP) (104-118) is a peptide inhibitor of PKC.